Korashy (2012) and fellow researchers published one of the first articles about the molecular mechanisms that govern the effect of camel milk on human cancer in 2012. Their research found the following:
- Camel milk inhibited HepG2 (liver carcinoma) and MCF7 (breast cancer) cells survival and proliferation through the activation of both the extrinsic and intrinsic apoptotic pathways.
- Camel milk significantly inhibited HepG2 and MCF7 cells proliferation (rapid increase in number).
- Camel milk contains lyzosomes, lactoferrins, immunoglobulins and higher amounts of antioxidant vitamins E and C, compared to bovine milk.
- Among these mediators, lactoferrin, an iron-binding glycoprotein, is known to exert in vitro and in vivo antitumor activity.
- This study demonstrated that camel milk induces apoptosis (a form of programmed cell death) in HepG2 and MCF7 cells through apoptotic- and oxidative stress-mediated mechanisms.